by GMFH Editing Team
We’re what our microbes do with what we eat. We’re pleased to share a new summary document on the latest evidence regarding the role of diet and probiotics on human health linked to their effects on the gut microbiota.
by GMFH Editing Team
GMFH continue to cover the important progress made in our knowledge of the gut microbiota's impact on wellbeing and disease.
Recent studies shed light on how early-life gut microbiota might help identify children at risk of developing obesity and T1D later in childhood.
by Stéphane Schneider
A new review explores mechanisms involved in gut microbiome modulation through exercise.
A scientific committee chaired has taken care to put together a program that covers the most relevant issues in the field today.
by GMFH Editing Team
Recent research has shed light on the importance of gut microbiota both during pregnancy and early life. Despite recent research that shows the placenta is not sterile, as previously thought, gut microbiota colonization in the first days and weeks after birth appears to have enormous significance for post-natal life.
by Andreu Prados
A recent study, led by Dr. Martin J. Blaser from the Departments of Medicine and Microbiology at New York University Langone Medical Centre in New York (USA), has found that early-life antibiotic exposure in non-obese diabetic mice may accelerate T1D development.
by Andreu Prados
It has been previously reported that the gut microbiota could be involved in the pathogenesis of metabolic diseases such as diabetes. A recent study, led by Dr. Wolfgang zu Castell from the Scientific Computing Research Unit at Helmholtz Zentrum München in Munich (Germany) has found that butyrate may have a protective effect in the pathogenesis of type 1 diabetes.
A long-term goal of gut microbiota research is to find a method of normalizing the microbiota early in life and sustaining it though the lifespan.
by Patrice D. Cani
In this paper, the first aim we had with Dr. Amandine Everard, was to investigate whether some key molecules involved in the innate immune system, mainly MyD88 [myeloid differentiation primary response gene 88], may contribute to the development of obesity, diabetes and low grade inflammation.
